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病理学-供医学各专业本科生.留学生.长学制.研究生双语教学使用

病理学-供医学各专业本科生.留学生.长学制.研究生双语教学使用

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  • ISBN:9787811164862
  • 装帧:暂无
  • 册数:暂无
  • 重量:暂无
  • 开本:16开
  • 页数:472
  • 出版时间:2009-08-01
  • 条形码:9787811164862 ; 978-7-81116-486-2

本书特色

《病理学(第8版)(英文改编版)》是专门为中国医学生打造的病理学双语教材。由国内20家医学院校的病理学教授联合美国华裔病理学者依据Robbins Basic Pathology的*新版(第8版)进行改编。改编以国内教学大纲为依据,对原版内容进行了删节、调整和适当补充。改编版教材既保留了原版精华,保证教材权威性,又能够*大限度地适应国内双语教学需求。供医学各专业本科生、留学生、长学制、研究生双语教学使用。

内容简介

简介   本书依据robbins basic pathology的*新版第8版进行改编。在改编过程中,对本书的内容进行了删节、调整和适当补充,个别章节有较大的更新和改动。同时,在内容上兼顾了临床医学及其他相关专业和不同学制的需求。因此本书可用作临床医学、口腔、公共卫生专业的五年制、七年制、八年制和留学生的双语教学用书,也可作为病理医生和进修生的重要参考书,以及作为执业医师资格考试的复习用书。

目录

Introduction to PathologyWhat is Pathology?How to Study Pathology?Diagnostic and Research Techniques in PathologyWhat Do Pathologists Do?The History and Evolution of PathologyPathology in ChinaChapter 1 Cell Injury, Cell Death, and AdaptationsCellular Adaptations to StressCell Injury and Cell DeathCellular AgingChapter 2 Tissue Repair: Regeneration, Healing, and FibrosisThe RegenerationThe Nature and Mechanisms of Action of Growth FactorsExtracellular Matrix and Cell-Matrix InteractionsCell and Tissue RegenerationRepair by Connective TissueCutaneous Wound HealingFracture Healing Pathologic Aspects of RepairChapter 3 Hemodynamic DisordersEdemaHyperemia and CongestionHemorrhageHemostasis and ThrombosisEmbolismInfarctionChapter 4 Acute and Chronic InflammationOverview of InflammationAcute InflammationChronic InflammationThe Possible Outcomes of InflammationThe Significance of the Inflammatory ResponseChapter 5 Diseases of the Immune SystemAutoimmune DiseasesRejection of TransplantsImmune Deficiency DiseasesChapter 6 NeoplasiaDefinition and General MorphologyDifferentiation and Anaplasia of TumorsGrowth and Spread of TumorsEffects of Tumor on HostIdentification Between Benign and Malignant TumorsNomenclature and Classification of TumorsBrief Introduction of Common NeoplasmsCarcinogenesis and Etiology of NeoplasiaHost Defense Against Tumors: Tumor ImmunityChapter 7 Genetic, Environmental and Nutritional DiseasesGenetic DiseasesMendelian Disorders (Diseases Caused by Single-Gene Defects)Disorders with Multifactorial InheritanceCytogenetic DisordersEnvironmental DiseasesEnvironmental Pollution and Occupational ExposuresAddictions and Associated DiseasesInjury by Physical AgentsNutritional DiseasesMalnutritionProtein-Energy Malnutrition (PEM)Vitamin DeficienciesObesityChapter 8 Cardiovascular SystemThe HeartIschemic Heart Disease (IHD)Hypertensive Heart DiseaseValvular Heart DiseaseCardiomyopathiesPericardial DiseaseThe Blood VesselsArteriosclerosisAtherosclerosisHypertensive Vascular DiseaseChapter 9 The LungPulmonary InfectionsObstructive Pulmonary DiseaseDiseases of Vascular OriginDiffuse Interstitial (Restrictive, Infiltrative) Lung DiseasesTumors of Respiratory SystemChapter 10 The Gastrointestinal TractEsophagusEsophagitisBarrett EsophagusEsophageal CarcinomaStomachGastritisGastric UlcerationGastric TumorsSmall and Large IntestinesInflammatory Bowel DiseaseTumors of the Small and Large IntestinesAppendix Acute AppendicitisTumors of the AppendixChapter 11 The Liver, Gallbladder, Biliary Tract, and PancreasThe LiverPattems of Hepatic InjuryViral HepatitisAutoimmune HepatitisDrug-induced Liver DiseaseAlcoholic Liver DiseaseMetabolic Liver DiseaseCirrhosisMalignant Tumors of The Liver OriginDisorders of the GallbladderCholelithiasis (Gallstones)CholecystitisCarcinoma of The GallbladderThe PancreasPancreatitisPancreatic CarcinomaChapter 12 The Hematopoietic and Lymphoid SystemsWhite Cell DisordersNon-Neoplastic Disorders of White CellsNeoplastic Proliferations of White CellsChapter 13 The Kidney and Its Collecting SystemClinical Manifestations of Renal DiseasesGlomerular DiseasesDiseases Affecting Tubules and InterstitiumTumors Chapter 14 The Male Genital SystemPenisScrotum, Testis, and EpididymisProstateChapter 15 The Female Genital System and BreastCervixCervicitisTumors of the CervixBody of UterusEndometriosisEndometrial HyperplasiaCbapter 16 The Endocrine SystemCbapter 17 The Musculoskeletal SystemChapter 18 The Nervous SystemChapter 19 Pathology of Infectious DiseasesChapter 20 Parasitosis
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节选

《病理学(第8版)(英文改编版)》依据Robbins Basic Pathology的*新版第8版进行改编。在改编过程中,对《病理学(第8版)(英文改编版)》的内容进行了删节、调整和适当补充,个别章节有较大的更新和改动。同时,在内容上兼顾了临床医学及其他相关专业和不同学制的需求。因此《病理学(第8版)(英文改编版)》可用作临床医学、口腔、公共卫生专业的五年制、七年制、八年制和留学生的双语教学用书,也可作为病理医生和进修生的重要参考书,以及作为执业医师资格考试的复习用书。

相关资料

插图:Intracellular AccumulationsUnder some circumstances cells may accumulate abnormalamounts of various substances, which may be harmless orassociated with varying degrees of injury. The substancemay be located in the cytoplasm, within organelles(typically lysosomes), or in the nucleus, and it may besynthesized by the affected cells or may be producedelsewhere.There are three main pathways of abnormal intracellularaccumulations:A normal substance is produced at a normal or anincreased rate, but the metabolic rate is inadequate toremove it. An example of this type of process is fattychange in the liver.A normal or an abnormal endogenous substanceaccumulates because of genetic or acquired defects inits folding, packaging, transport, or secretion. Mutationsthat cause defective folding and transport may lead to accumulation of proteins (e.g., l-antitrypsin deficiency).An abnormal exogenous substance is deposited andaccumulates because the cell has neither the enzymaticmachinery to degrade the substance nor the ability totransport it to other sites. Accumulations of carbon or silica particles are examples of this type of alteration.Fatty Change (Steatosis). Fatty change refers to anyabnormal accumulation of triglycerides within paren-chymal cells. It is most often seen in the liver, since this isthe major organ involved in fat metabolism, but it may alsooccur in heart, skeletal muscle, kidney, and other organs.Hepatic steatosis may be caused by toxins, proteinmalnutrition, diabetes mellitus, obesity, and anoxia.Alcohol abuse and diabetes associated with obesity arethe most common causes of fatty change in the liver (fattyliver) in industrialized nations.Free fatty acids from adipose tissue or ingested foodare normally transported into hepatocytes. Excessaccumulation of triglycerides may result from defectsat any step fro

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